We Cured Chronic Prostatitis (most people think it's impossible)

We Cured Chronic Prostatitis (most people think it's impossible)

We Cured Chronic Prostatitis (most people think it's impossible)brenden henry
Published on: 24/10/2024

We Cured Chronic Prostatitis (most people think it's impossible) He spent over $100,000 trying to fix chronic prostatitis and went to over 20 doctors, yet nothing worked. Imagine dealing with constant lower urinary tract pain, inability to have sex, urinating all over yourself, and the depression that comes with it. Many guys are driven to suicide because of this issue, and modern medicine seems to have no cure. But in this video, I'm going to show you how we do the impossible, and cure it for our clients with absolutely zero side effects. This is important because it can save you from the pain, the suicide, or prostate surgery. And I'm going to show you the scientific proof driving everything that we do. This is game-changing and life-changing, and people need to know about this. So we're not going to hold anything back. But before we dive in, I want to address something that's very important. This is something traditional medicine would have you believe is impossible, as most medical doctors don't research shit, and just follow what they're taught in institutions, who are working in concert with the pharmaceutical industrial complex. But the reality is, the medical system is failing people who are dealing with issues like chronic prostatitis, and we have to go beyond mainstream methods and outdated science. So let's dive in. Chronic non-bacterial prostatitis, or benign prosthetic hyperplasia, can be hell for people who have it, because they're facing with constant pain, inability or reduced ability to ejaculate, so they can't have sex, uncontrollable urination, feelings of incomplete bladder emptying, and urgency. And these are just some of the symptoms that can wear on people mentally, and they often suffer from depression, poor mood, which can sabotage relationships with friends, not just their significant other, due to an inability to perform sexually. Now, the mainstream offers almost no hope to these people. Now, this enlargement of the prostate, coupled with changes in the collagen around the urethra, and increased urethral length, results in increased urinary resistance and obstruction. A healthy weight of the prostate is about 20 grams, but even minor BPH is classified as 30 grams, and this is where minor symptoms start to develop. So it really doesn't take much, and once it reaches 50 grams, you're looking at an increased risk of acute urinary retention, and if this happens, surgery would be needed to remove the prostate. And then you lose the ability to ejaculate, and you may even need a penile implant just to achieve erection again. Now, the Forth Valley study in Scotland found that 14% of men aged 40 to 50 years old have BPH, and that increases to 43% among men older than 60. BPH has also been shown to be nearly as prevalent as hypertension and diabetes among patients who are seeking treatment for erectile dysfunction. So it's one of the leading causes, and you know it if this is the cause in the early stages, as you may not have much of an obstruction, but just have a reduced sexual drive, which you may think is coming from a hormonal imbalance, leading you down a path of trying to correct symptoms, but going the entirely wrong direction. Now, traditional treatments for benign prostatic hyperplasia often focus on alpha blockers, such as Tamsulosin or Alfuzosin, which can help to relax the smooth muscles in the prostate and bladder and help to accelerate the urine stream, or tadalafil, which works similarly, albeit through a different mechanism, which I made the most detailed video of on the entire internet, by the way. And in extreme cases, 5-alpha-reductase inhibitors like finasteride or dutasteride are used. However, these last ones work by reducing the conversion of testosterone into a more androgenic hormone known as DHT. And this can cause side effects such as reduced sense of masculinity, worse in mental state due to disruptions in the neurosteroid pathways, and decreased insulin sensitivity, which could contribute to diabetes. Now, they are effective at slowing prostate growth from androgen sensitivity and treat in certain types of hair loss like androgenic alopecia, but the side effects can be concerning, and the results are still not as good as what I'm going to cover in this video. One of our clients had been using Alfuzosin, an alpha blocker, and tadalafil, and he experienced some relief, but it wasn't enough. After starting prostatilen combined with our other synergistic peptide strategies, he saw a complete resolution of all symptoms, as if the problem had never even existed. Prostatilen contains peptides from the animal prostate tissue, and it's been shown in the literature to perform the impossible, by healing conditions deemed incurable. Now, despite these proven results, these peptides remain underutilized and not widely known. Some people dealing with chronic prostatitis fail to get relief from traditional medical treatments, even after going to urologists who specialize in these types of disorders. Recommendations such as prostatic massage, whether internal with a vibrator or external through high-powered shockwave therapy, often fail to provide adequate relief. And on YouTube, you can find numerous accounts of people enduring prostatitis for years, and the rate of suicide among those with prostatitis is 1.58 times higher compared to those without the condition. So in this video, I'm going to show you how prostatilen is not just superior to 5-alpha reductase inhibitors and alpha blockers, but how it also has absolutely zero side effects. This is important because it can save you from the pain, the suicide, or prostate surgery, which results in nerve damage, and in a high number of cases, men being unable to ever get an erection again without an implant, and also suffering urinary incontinence. While pelvic floor exercises can help if you're at that stage, and are things I recommend in general for male health, it would be much better that you avoid ending up in such a dire situation in the first place. So prostatilen improves blood flow to the prostate, bladder, and the kidneys, and it has a profound anti-inflammatory effect. It's been shown to reduce protein levels in the urine, and the phosphatase activity, and it also reduces CRP in the blood serum. Now in cases of benign prostatic hyperplasia, it decreases prostate weight, improves urination, and erectile function, and it's been shown to be more effective than 5-alpha reductase inhibitors and alpha blockers at improving symptoms. And it works long term too, through reducing the actual size of the prostate. That's right, something traditional medicine would have you believe is impossible. Unfortunately, many doctors don't research beyond what they're taught in institutions that often align with the pharmaceutical industrial complex. Prostatilen also demonstrated a 32.5% increase in sperm concentration per milliliter of ejaculate, and it improved sperm motility by up to 62% in the long term, and it decreased anti-sperm antibodies, reactive oxygen species, and leukocytes in the ejaculate. Testosterone levels and erectile function improved, and the catecholamines increased in the testes. Now this can boost your fertility and hormones. Now on top of that, prostatilen reduces blood serum triglyceride levels, uric acid, and platelet aggregation, while boosting immune function by increasing the CD4 count, which can even be beneficial in cases of infection in theory. Now these benefits are truly extraordinary, and can quickly give men their lives back after suffering from this debilitating condition for so long. But that's only if they're used correctly. And in my peptide mastery course, I cover the best route of administration, dosing, and how to stack prostatilen with synergistic peptides for even more effective results. This is important because if left untreated, it could potentially lead to prostate cancer, which is the second leading cause of cancer death in American men. Now while it's rarely diagnosed before the age of 40, lesions can begin to develop as early as the age of 30, and a rapid increase in tumor weight can occur thereafter. And that raises the risk of prostate cancer later in life. So it makes preventative treatment with prostatilen a smart idea. Now I want to briefly touch on another major cause of prostatitis, which is chronic bacterial infection. This is notoriously difficult to treat because antibiotics often struggle to penetrate the prostate, and there are these superbugs which are becoming more prevalent. And these can form these protective biofilms which shield them from antibiotics. And it's even worsened by quorum sensing, which is when these bacteria communicate through signaling molecules to coordinate defenses against antibiotics. Fortunately, there are strategies to disrupt this quorum sensing and improve antibiotic effectiveness, but they must be used in combination with the right antibiotics for each specific case. Now immune health also plays a critical role in the success of these treatments, because if you don't have a strong immune system, antibiotics just don't work as effectively. Thankfully, we have solutions to boost immune function and improve treatment outcomes as well. Now all of these protocols are thoroughly covered in my peptide mastery course, and we also offer personalized coaching through our molecular bioenhancement coaching programs.

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Why this Nutrition PHD is WRONG: Seed Oils Accelerate Aging

Why this Nutrition PHD is WRONG: Seed Oils Accelerate Aging

Why this Nutrition PHD is WRONG: Seed Oils Accelerate Agingbrenden henry
Published on: 10/10/2024

Our mitochondrial functioning is a huge predictor of our overall health and longevity. For example, they power each cell in our bodies and our cells make up every single tissue in our bodies. And when our mitochondrial functioning goes down, we can see a reduction in VO2 max, which is associated with a worse longevity outcome in cardiovascular disease, and we see a higher rate of biological aging according to DNA methylation tests. And so what we consume on a daily basis contributes significantly to our overall mitochondrial functioning. And one significant aspect of our diet is fatty acids. So the type of fats we consume really do matter because they can actually end up as part of cardiolipin, which is a phospholipid on the inner mitochondrial membrane, which is the same place where the electron transport chain exists, which produces ATP, which powers your cells. And so when the wrong type of fatty acids, such as polyunsaturated fatty acids, become a part of this cardiolipin, or they make up more of cardiolipin than other types of fats, it's a problem because polyunsaturated fatty acids are more prone to oxidation in the body, and this has to do with them having more double carbon bonds. And so they can cause mitochondrial stress and overall reduce the cellular functioning. It's been proven on a mechanistic data that polyunsaturated fatty acids can raise MDA levels, which is a marker of oxidative stress, and they can reduce antioxidant defenses. And so this is really important because when you actually look at another peptide known as SS31, this peptide has been shown to bind to cardiolipin, and it improves the overall mitochondrial functioning. It improves the heart function because, as you know, our heart actually has more mitochondria than any other organ in our body. And so this improves our cardiovascular function, and hence our VO2 max, and it's associated with a longer longevity. So the problem I have here is that, first of all, Lane Norton made a video where he was mentioning that seed oils actually are good according to a randomized controlled trial. Well, I want to tell you something about randomized controlled trials. First of all, the two longest ones ever conducted in the history of our world so far, Sydney Coronary Experiment, which was six years, it's a randomized controlled trial, and the Minnesota Coronary Experiment, which was four years. Now, these were well-controlled studies, and they evaluated polyunsaturated fatty acids consumption versus saturated fatty acids consumption in another group. And the main outcome of both of these studies was the same. There was an increased rate of cardiovascular-related deaths in the polyunsaturated fatty acid group, and there was an overall increased mortality in the polyunsaturated fatty acid group. Now, some confusion arises here, because it's also been shown that the saturated fat group does have a higher LDL level of cholesterol. This is because polyunsaturated fatty acids do inhibit cholesterol synthesis to a degree, but despite that fact, they can't offset all the other damaging effects that come from the oxidation on cardiolipin and impaired mitochondrial and cellular functioning, which has devastating effects on our entire body. And so, in this video, I want to go over what Lane Norton was saying, because, you know, he was basically saying that mechanistic data doesn't mean anything, we've got to look at randomized controlled trials. And I agree to an extent, but let me tell you why. I'll tell you a story. So, a while back, I've had people message me in regards to one specific mechanism that a peptide has, and they're concerned over this one mechanism causing an issue, such as, for example, pinealon has been shown to upregulate GDF11. High levels of GDF11 have been associated with liver fibrosis, so people were worried that pinealon would cause liver fibrosis. Well, the problem is, it's been shown that rapamycin, through induction of autophagy, can prevent liver fibrosis while GDF11 treatment is administered. Well, pinealon has other mechanisms. For example, it upregulates irisin. Irisin is a myokine, which induces autophagy in the liver, so it would counteract this, okay? And you can confirm it, too, by actually looking at the long-term human studies or animal studies, which show absolutely no signs of liver issues on imaging over a six month period of megadosing it, okay? Or let me give you another example. Epitalon. Some people like to say that, okay, because it upregulates TERT gene expression, which is telomerase, that it could be worse for cancer. Well, the problem with that is that epitalon has tons of mechanisms downstream, which are literally anti-cancer and inhibit tumor growth. Let me give you another example. Because even neuroscientists can fall victim to this looking at only mechanistic data trap, so I'm not saying that Lane Norton is completely wrong by what he says here. He raises a good point. So Andrew Huberman was talking about how BPC-157, through increasing VEGF, may contribute to cancer. Okay, but, you know, exercise increases VEGF, but the blood from people who exercise has actually been shown to inhibit cancer growth. Or let me give you another example. Hyperbaric oxygen chamber therapy. This increases VEGF as well, but it's also been shown to inhibit cancer growth. And so, I promise I'm getting to a point here. When Lane Norton said, only look at randomized controlled trials, and not just the individual mechanistic data, the only problem I have with this is that randomized controlled trials often control for the variables very poorly. And a good example of this is the Identical Twin Study, which was released sometime early this year, or maybe last year, where they put one identical twin on a vegan diet, and the other identical twin on an omnivore diet. And they were evaluating different health markers, but also ones associated with longevity. So they looked at the DNA methylation tests, and also the telomere length. And the outcome that they found was that the vegan group had better markers overall, a slower rate of aging. So a lot of people were quick to jump to the conclusion that, okay, this settles it, you know, veganism is the best, and omnivore is bad for longevity. And that's what a lot of people say, who just read like the title of a randomized controlled trial, maybe the abstract, and then the conclusion. But when you look at the actual details, guys, you'll see that despite going through all the effort to bring in identical twins to this study, they fail to control for variables such as keeping the carbohydrate source between both groups consistent. Oh no, you have the omnivore group getting white rice, which is vastly insuperior to the quinoa, which the vegan group was getting. Okay, quinoa is much more nutrient dense, it doesn't have arsenic like rice does, but that's just one example, guys. They're eating out at restaurants, and the omnivore group was rating a higher satisfaction to the food at the restaurants. This could be assumed that he could possibly be just ordering worse quality foods, which taste better, and they're probably cooked in seed oils and stuff at restaurants, because that's what they usually use. And so this study is basically garbage, guys. I mean, if you're not going to control for at least the most important variables in a study and have the only difference be the actual protein source, then what's the point of even doing the study? And not to mention, you know, some of these randomized controlled trials, especially in nutrition science, are tricky because they may only be like six months long, maybe one year, but did you know linoleic acid has a half-life in the body of two years? So when it becomes part of cardiolipin and accumulates in your adipose tissue, it can actually stay there for quite a long time, guys. That's why the studies that I was looking at were six years and four years, respectively, for the Sydney Coronary Experiment and the Minnesota Coronary Experiment. So anyway, I hope this sums things up. In regards to Lane Norton, I'd like him to actually pay a lot of attention to these randomized controlled trials, because when you look at the details, some things just don't add up. And I do think that looking at the mechanistic data in these cases is very important to confirm findings in these trials. So that's basically all I have to say on that. I hope that you avoid seed oils. Don't fall into the scandal. Extra virgin olive oil, high in polyphenols, is great. And Brian Johnson's one is actually pretty good. Despite me just making a video reviewing Brian Johnson's blueprint supplement stack and overall not recommending it due to just two main problems with it, go check out the video if you're interested in that, I do think that extra virgin olive oil he has is really good. It's high in polyphenols, and I have no problem with it. It's usually single source. It even lists the source on it, and the testing data looks great. And if you're interested in cardiovascular health, you'll probably want to be using the vesugen peptide. And this peptide has been shown to reverse the senescence-associated secretory phenotype in blood vessels. It's been shown to restore blood vessel function, lower LDL levels, and even reverse markers of biological aging. So if you haven't seen the video on my channel where I talk about vesugen, you should go check that out. But that's it. And also, get your free Life-Changing Magic of Peptides course on my website, link in the description. It'll help you learn more about peptides and how they can be used to improve your health and longevity. This has been Brendan Henry. People call me the world's leading expert in peptide science, and I hope this has been helpful.

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Secrets of Tadalafil

Secrets of Tadalafil

Secrets of Tadalafilbrenden henry
Published on: 09/10/2024

What if I told you that Cialis, a drug known for its use by elderly men to strengthen their erections, is actually one of the most potent health enhancers for men on the planet? Most guys are embarrassed at the thought of even using it, but Tadalafil, as it's officially called, is not just erectile dysfunction medication. Not only does it help with blood flow for erections, but this ED medication can literally prolong stem cell life, which can give your body the ability to regenerate almost any tissue, even if they normally won't heal. For example, one guy had a foot wound that would not heal for 20 years until he started Tadalafil. Tadalafil is useful for improving the function of the heart after an ischaemic injury such as a heart attack, and has been shown to result in a 13% lower incidence of major cardiovascular events, 25% lower mortality, and 39% lower cardiovascular related mortality compared to a control group in patients who had at least one cardiovascular disease risk factor. Tadalafil also enhances muscle growth and enhances bone strength through its ability to increase testosterone and upregulate androgen receptors. Tadalafil helps to reduce symptoms of benign prostatic hyperplasia by improving the testosterone to estrogen ratio, relaxing smooth muscle in the prostate and bladder, and improving blood flow, which can reduce symptoms like frequent urination and slow urination. Tadalafil can reduce blood pressure and improve endothelial function, thus lowering the risk of cardiovascular disease through regulating endogenous antioxidant production and dilating arteries, which can help prevent a blood clot from forming. Many people take aspirin to help prevent blood clots, and this works by thinning the blood, but the downside to this is that it can increase brain bleeds from trauma. Tadalafil doesn't thin the blood, it just dilates arteries, which could potentially make it a safer option, but it can still cause side effects if you use it incorrectly. So by the end of this video you will learn how to use Tadalafil most effectively, and an incredibly powerful synergistic combination you can use with it to get more healing power. My name is Brenden Henry, the world's leading expert in peptide science. I was the first person to make a video on the incredible power of the vesugen peptide for enhancing the proliferation and differentiation of mesenchymal stem cells and delaying their senescence. This is huge because stem cells cost tens of thousands of dollars, but this peptide can increase your own naturally for a small fraction of the price of stem cell clinics. This can lead to the acceleration of healing in your body. Whether you have an organ that's damaged, a joint, muscle, or recently undergone a surgical procedure, this will help you heal more quickly. And you can get even more benefits out of it by combining it with Tadalafil. This protocol can work well on its own, or it can be used to enhance the effects of stem cells you receive at the clinic. I currently have a client who purchased my peptide mastery course and coaching, link in the description, and he is looking to maximize the potential of a stem cell treatment at the clinic. So this is of course one of the things included in our protocol, and just one example of how traditional medications or supplements can synergize with certain peptides in ways that are not known by the majority of the people on the planet. In this video, I want to talk about what Tadalafil is and how it works on a mechanistic level so that you can understand the benefits it has to offer more clearly and be more willing to confidently use it to enhance yourself. Then I will tell you how to best use it and dose it so that you get all of the benefits without any toxic effects. First, Tadalafil is a PDE5 inhibitor that prevents the breakdown of CGMP, which enhances blood flow by promoting vasodilation. While it doesn't create CGMP directly, which requires nitric oxide, it helps increase CGMP levels by stopping its degradation. However, PDE5 inhibitors have some crosstalk with PDE8, which are high affinity C-AMP specific enzymes, leading to increased nitric oxide production indirectly. This crosstalk is significant because without it, these inhibitors would be much less effective because they'd have less nitric oxide to work with. Tadalafil's attributes extend to stimulating endogenous antioxidant production in muscle cells, cavernous tissue, and lung endothelial cells. This effect is due to hormesis, which is the body's upregulated production of endogenous antioxidants in response to a mild stressor, leading to a net protective effect. Evidence of this is seen in lung endothelial cells, where Tadalafil, by activating the CGMP-PKG1 pathway, enhances cellular antioxidant defenses, protecting against oxidative stress and dose damage. This leads to improved endothelial function in humans. Tadalafil also reduces the risk of thromboembolism, which is blood clots in males. Getting further into the mechanistic data, PDE5 inhibition increases CGMP levels, which stimulates PKG1 and also leads to an increase in androgens. PKG1 and PKG2 mediate the effects of CGMP, and Leydig cells express mRNA for both. However, inhibiting PKG leads to a dose-dependent decrease in androgen production, indicating that PKG is necessary for the CGMP-driven rise in androgens. When Tadalafil activates CGMP-PKG1, it phosphorylates the star protein, enhancing the transport of cholesterol into mitochondria and increasing testosterone production in Leydig cells. PDE5 inhibitors also interact with PDE8, leading to an intracellular C-AMP increase in Leydig cells, further facilitating testosterone production. Mice with PDE8 knockout have up to 4x more testosterone. Tadalafil has been shown to increase testosterone levels by an impressive 40% in men with metabolic syndrome over time. In a direct comparison with Sildenafil, Tadalafil has been found to be superior at increasing testosterone. Tadalafil can also enhance the testosterone-to-estrogen ratio by inhibiting the aromatase enzyme, and it increases androgen receptor density while improving lean body mass. It has been shown in human studies that you can expect to gain a couple pounds of lean mass and lose a couple pounds of fat from long-term Tadalafil use. Tadalafil improves bone strength and wound healing. It increases androgen receptors and decreases aromatase in osteoblast cell lines and was able to enhance fracture healing in a rat femur and improve bone density. Tadalafil also improves burn wound healing. Moving up to the brain, in elderly patients with erectile dysfunction, Tadalafil improves cerebral blood flow in the postcentral gyrus, precuneus, and brainstem, and enhances cognitive function, as assessed by the Montreal Cognitive Assessment Score. While it's uncertain if this effect occurs in younger men, Tadalafil has enhanced working memory in both young and old mice. Now let's get into how you should dose Tadalafil for maximum benefits and low side effects, even though its safety is well established. If you dose it too high, you may get occasional headaches, flushing, a stuffy nose, but there are two even worse side effects than that. One, muscle pain. Two, aortic damage and testicular toxicity. The muscle pain can occur because Tadalafil is a slight off target for PDE11 inhibition, which is known to cause muscle pain, especially back pain. At 5mg, this effect of Tadalafil is barely relevant, but it becomes more relevant once you go up to 10mg+. The aortic damage can occur due to oxidative stress combined with too much dilation. You see, Tadalafil is a hormetic stressor, as mentioned earlier, and this is how it increases endogenous antioxidant production, and is consistently shown to be net protective at 5mg human dosages. It's protective to the testes at these doses, as well, in both humans and mice who received an equivalent dose converted for their species. However, if you look at rodent studies, you will see that at a 20mg equivalent dose, that changes, and starts to have a net toxic effect. MDA levels increase, which are markers of oxidative stress, and antioxidant levels drop. The same case would be true if you did HBOT for 15 hours a week, which is hyperbaric oxygen chamber therapy. Some is great, but once you cross a certain threshold, it becomes toxic. This also relates to how some people using Tadalafil mention a slight dip in sex drive over time. This can occur if they are taking too high of a dose, usually 10mg+, and it can happen for a couple reasons. The first, is that it can start to induce oxidative stress in some people, as mentioned. And the second, is that it can reduce estrogen levels with long-term use, and estrogen is important for libido. This is why you need to use no more than 5mg a day, and also monitor your blood work for your testosterone to estrogen ratio. But you need to request LC-MS, MS, because this is the only accurate way to measure hormone levels. Doctors are mostly just concerned if something is within range, not if it's optimal. If your estradiol levels are near the low end of the range, and your testosterone levels are mid-range or high range, then you may consider adding in something to raise your estradiol up. DHEA or Omnibol, which contains pregnenolone and DHEA, is great for this, and Boron can help some people to a lesser extent, and also raise free testosterone slightly. In conclusion, Tadalafil is a fascinating and underappreciated compound with uses in healing broken bones, chronic wounds, heart damage, and increasing testosterone levels. For the best results, for both blood flow and stem cells, consider combining it with Vesugen, a tripeptide with a sequence lys-glu asked. To learn more, get the peptide mastery free edition on my website, linked in the description. You can also get our free guide, Toxic Testosterone, which reveals the hidden dangers of the 5 most commonly recommended testosterone supplements, and what to do instead. This has been Brenden Henry, the world's leading expert in peptide science.

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Fitness Industry: Not So Fit

Fitness Industry: Not So Fit

Fitness Industry: Not So Fitbrenden henry
Published on: 23/04/2024

Scrolling through my YouTube feed reveals numerous thumbnails featuring some of the most popular fitness YouTubers indulging in processed and fast foods. Some have even created brands selling these foods, filled with poor ingredients, while advoc

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